Big fit horses in reasonable work can get laminitis too…

Laminitis is not just a disease for small natives: big, fit horses in medium work can get laminitis too, as I discovered to my chagrin a few weeks ago.

I was chatting about our recent troubles at the area 20 qualifiers yesterday and the lady I was chatting too said “Oh, he’s a big horse, we forget they can get laminitis too” as if this was rare?

It had never occurred to me that laminitis was mainly a disease of small ponies, although I do associate it mostly with good doers. Cal is a good doer, but he is also a big, fit horse in medium level work and had been eventing the week before he showed that big fit horses in reasonable work can get laminitis too.

The causes of laminitis are now known to be metabolic, either associated with Equine Metabolic Syndrome ( a sort of type II Diabetes for horses) or Equine PID, more commonly known as Cushing’s disease. Metabolic causes means that laminitis is a disease of the whole horse, the cause and the treatment are not limited to the foot.

I’m pretty sure Cal has EMS, although I’ve never tested him properly. How do I know this? Because he has been such a tricky barefooter over the years. For those of you who do not fully embrace the barefoot concept, let me share with you my paradigm.

Any horse with the correct diet, environment, exercise and trim should be able to go barefoot and work hard barefoot.

Those 4 simple sounding words are not simple things to achieve in the U.K. Cal is an Irish Spirts horse, so he is half Irish Draught, and he looks like he got quite a lot of Connemara in the mix, so a dose of Spanish blood too. He didn’t get much TB in his phenotype, that’s for sure.

Diet-  Cal is finely tuned to survive in the Irish peat bogs, or possibly also in Spanish scrubland. He doesn’t get much green grass, a sniff makes him footsore so a good bellyfull would probably kill him. He is the main reason our horses are track dwellers, and his story is partly why we bought our house and land, because traditional livery yards simply could not cater for his needs. This horse loves fresh thistles, bashes down nettles to let them wilt, eats a bit of bracken for the insulin like compound, goes for ivy, again for the sugar busting properties. He is pretty good at managing his own condition, as long as he is offered the variety of herbs and plants he needs to offset the green posion. He gets a small bucket feed which contains salt, Phytorigin GI, a hindgut balancer ained at feeding the good bacteria, Phytolean plus, a plant based supplement with lots of anti-oxidants designed to support the immune system and homeostasis of tricky metabolic horses.

Environment: he on a track system or paddock paradise. His main needs #friendsforagefreedom are met as best we can. He lives out 24/7, in a stable herd with his mates, to groom, play, commune with and boss around. They have access to constant ad lib forage, and are safe from stress. As he is pretty dominant he is the safest of all from stress, especially as Paddy is the lookout.

Exercise: he’s my main horse. He lives on a track so does about 5 miles a day mooching around on there, he also gets ridden 3-4 times a week, a mixture of hacking, schooling, jumping and fast work every 10 days or so. Of course he could do more, if I had more time.

Trim: trim has always been tricky. But that’s mainly because Cal has been tricky. The more I learn about feet, the more I think there difference between a good trim and a bad trim is a bit like a clip: two weeks!! Bad feet are impossible to trim into a healthy shape and function, and good healthy working feet are really hard to trim into bad shape because they just wear themselves correct again with work and movement. Cal has been footy on stones for his entire barefoot career. We use nice little euphemisms but make no mistake, a slightly sore foot is a slightly weak or a pathological foot. That’s why I would never call a horse sound unless it was truly sound without shoes: if the horse is sore when you take the shoes off, the shoes are disguising a problem. It took me a few years of looking at hoof photos to realise that Cal was a sub-clinical laminitic.

When I bought him his feet ran so far forward the whole foot sat in front on his legs, but he was sound as a pound in shoes! When he broke his carpal bone and we took the shoes off it took 3 full years to get a hoof that actually had hoof under the leg bones, and 4 years to get the heel bulbs in line with the middle of his cannon bones. The under run heels, the slipper like toes, the occasional growth ring, these were all subtle laminitic stigmata. Yet he had worked hard, team chased, hunted, evented, with the only sign of challenge being on very stony ground. So many people said I should just shoe him, as if that would solve all our problems, and that advice even came from some barefoot trimmers and vets.

Had he been shod, I might not have spotted the mild attack of laminitis until it was a full blown disaster.

I had brought him down to the house ready to compete at the weekend. I had ridden him in the school, bathed, cleaned tack and left him in the stable at the house for an early start. Normally when at the house they get Horsehage HiFi Haylage,

but our local shop had run out so I had bought some West Lancs Haylage instead. I gave him a good feed and a good big section of Haylage to last him overnight. The next day he was pointing a foot at me, and shifting around behind.

It took me a few days to twig what was going on: because one foot seemed to be worse I thought abscess first of all. And I was still feeding the West Lancs Haylage. It was only when I realised it was pure Ryegrass Haylage that I put two and two together. After a few days at the house no abscess had appeared and he wasn’t actually a welfare case so I moved him back to the field. He got better there but after 10days was still not looking rideable. He had palpable pulses in all 4 legs and was moving very slowly and appeared miserable.

I got the vet out, who agreed with me that it was laminitis, but very mild, to the extent that, I quote, “a lot of owners wouldn’t have noticed there was anything wrong”. He gave Cal a shot of i.v. analgesia which allowed me to get hoof boots on his front feet so he was comfortable enough to walk back to the house, and then to march him up the big hill. I kept him at the house, rationing every mouthful: no grass at all, a section of Hifi or a tiny feed very 4 hours and walking up the hill once or twice daily. All this strict diet and exercise was aimed to sharpen his insulin response again. He had Phytorigins Rescue Remedy which is a 5 day course, double dose PhytoGI, double dose Phytolean Plus for maximum antioxidants and a sachet Danilone twice daily.

http://phytorigins.co.uk/Phyto-Rescue-Remedy

After 4 days he was much improved, back to hacking out and schooling again at 10days. He went back to the now very dry sandy grass free track (thanks weather) on about day 5 (more to do with work than precise symptoms).

The vet offered to do a glucose stimulation test to see if it was definitely EMS- I have declined this. The blood test says it’s not Cushings, there is no really effective treatment for EMS other than really tight management which we do already, and there is a significant risk of laminitis from the stimulation test.

I now know that every mouthful counts, that I will never switch Haylage again for my own convenience, and that this horse needs to work every week, no matter how busy I am with my job.

It’s been a bad spring. I have another medical friend whose horse got laminitis because she was a bit busy with work and didn’t ride for a week: nothing else changed. And I have heard local tales of other big, fit horses in reasonable work who have succombed to the condition after a seemingly innocent change in diet or management. The grass this spring has been bonkers, wet and warm and then sunny is a great combination for really rich Cheshire cow grass. Our track looks totally bare now but it’s the scorching sun that has killed the green stuff the last couple of weeks, before that it was the horses munching away that kept the grass looking poor.

Do you check your horse’s pulses every day?

http://www.ironfreehoof.com/equine-digital-pulses.html

Shod or not, a palpable pulse might be the first sign of impending laminitis and feeling a change early might just save your horse from a full blown attack.

https://thehorse.com/111374/10-early-warning-signs-of-laminitis/

Do you watch every mouthful your horse eats?

Keeping a tricky barefoot horse sound, healthy and in full work is a sure way to turn into a feed geek; Paddy could eat more or less what he liked and still trot and canter on any stony surface in the forest.

Since having Cal my rudimentary knowledge of horse physiology and nutrition is now more or less at degree level; of course it helps that I am already an expert in human physiology so the proper equine textbooks are legible to me. I have tried every supplement on the market, tried every supposedly healthy bagged feed and have come around to the acceptance that maintaining a healthy hindgut is key, and that all is really required is hay, water, salt and enough variety in their environment to allow them to forage for what they need. in the absence of variety, supplements might be required and it’s the Phytorigins approach that makes the most sense to the cynical scientist in me.

Do you reduce the bucket feed if your horse is doing less work?

Cal isn’t on anything rich or high in protein or sugar, we use Agrobs, but I have cut down significantly from what I was feeding and will cut down even more if he has a quiet week. He wasn’t fat, but his condition hasn’t really changed on less food so I think feeding the minimum required to keep him fit is definitely the way to go. Even in a busy month, he will never be in hard work like a polo pony or a racehorse.

Cal fully recovered at BRC area qualifiers